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Mazetol (Acetazolamide)
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Mazetol

Mazetol is a carbonic anhydrase inhibitor that affects your body's fluid balance. It helps remove water and electrolytes in your urine, lowers the amount of fluid in your eyes. It calms your nervous system, and allows your body to use oxygen better at high altitudes.

Other names for this medication:
Abzole, Aceact, Acetamide, Acetamin, Acetariv, Acetazolamide, Actamid, Actamide, Actazid, Antilep, Axytex, Carbadac, Iopar, Mazetol, Mezapin, Synomax, Tegrital, Zolamide

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Also known as: Acetazolamide

Description

Mazetol contains an active ingredient Acetazolamide, which belongs to class of drugs called carbonic anhydrase inhibitors. Mazetol effectively treats certain types of glaucoma (excessive pressure in the eyes) by reducing the amount of fluid in the eye, and thereby decreases pressure inside the eye. Acetazolamide acts also as a diuretic ("water pill") and inhibits the protein in the body called carbonic anhydrase. This leads to reducing the build-up of certain fluids in the body, significantly alleviating the symptoms of congestive heart failure. Acetazolamide is also used to treat certain types of seizures, and to treat or prevent altitude sickness.

Dosage

Follow all directions on your prescription label. Your doctor may occasionally change your dose to make sure you get the best results. Do not use Mazetol Sequels in larger or smaller amounts or for longer than recommended.

Your dose of this medicine will depend on the condition you are treating. If you take Mazetol Sequels for congestive heart failure, your doctor may tell you to skip your medication for a day. Follow your doctor's dosing instructions very carefully.

Take this medicine with a full glass of water.

While using Mazetol Sequels, you may need frequent blood tests.

Mazetol Sequels may be only part of a complete treatment program that may also include other medications. Follow your doctor's instructions very closely.

Store at room temperature away from moisture and heat. Keep the bottle tightly closed when not in use.

Detailed Mazetol Sequels dosage information

Overdose

Seek emergency medical attention or call the Poison Help line at 1-800-222-1222.

Storage

Store at room temperature between 20 and 25 degrees C (68 and 77 degrees F) away from moisture and heat. Throw away any unused medicine after the expiration date. Keep out of the reach of children.

Side effects

The most common side effects associated with Mazetol are:

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Side effect occurrence does not only depend on medication you are taking, but also on your overall health and other factors.

Contraindications

Mazetol may impair your thinking or reactions. Be careful if you drive or do anything that requires you to be alert.

Avoid exposure to sunlight or tanning beds. Mazetol can make you sunburn more easily. Wear protective clothing and use sunscreen (SPF 30 or higher) when you are outdoors.

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No-slip hydrodynamic boundary condition for hydrophilic particles. This article examines the existing literature on the links between glycemic control, arterial hypertension, high cholesterol and hyperlipidemia, obesity, inflammatory markers, sleep-disordered breathing, and exercise with risk of diabetic retinopathy development and prevention. Diagnostic performance of quantitative kappa and lambda free light chain assays in clinical practice. Measuring shared decision making processes in psychiatry: skills versus patient satisfaction. Benzodiazepine binding was decreased in the cortex and hippocampus at day 7 compared to day 1 of lorazepam, with an increase in CL 218,872-resistant (Type 2) sites in both regions. The effectiveness of intravesical electrostimulation (IVES) in the treatment of acute prolonged bladder overdistension (PBO) was investigated. After 400 generations, the best agent recognized imminent collisions reliably in the familiar environment where it had evolved. The findings show that a high-fibre diet modulates plasma lipid, insulin and glucose levels and modifies the plasma lipoprotein distribution and composition in normolipaemic, lean rats as well as in hyperlipaemic, genetically obese rats. Thus, unlike in previous studies in the hippocampus, enhanced or decreased PSD-95 clustering alone was insufficient to drive AMPA or NMDA receptors into or out of SPN synapses.

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